<?xml version='1.0' encoding='utf-8'?>
<oai_dc:dc xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:oai_dc="http://www.openarchives.org/OAI/2.0/oai_dc/" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xsi:schemaLocation="http://www.openarchives.org/OAI/2.0/oai_dc/ http://www.openarchives.org/OAI/2.0/oai_dc.xsd">
  <dc:contributor>W. James Fleming</dc:contributor>
  <dc:contributor>H. C. Murray</dc:contributor>
  <dc:creator>W. C. Eastin Jr.</dc:creator>
  <dc:date>1982</dc:date>
  <dc:description>&lt;p&gt;&lt;/p&gt;&lt;p&gt;1. Adult black ducks (&lt;i&gt;Anas rubripes&lt;/i&gt;) were given freshwater or saltwater (1.5% NaCl) for 11 days and half of each group was also given an organophosphate (17 p.p.m. fenthion) in the diet on days 6–11.&lt;/p&gt;&lt;p&gt;2. After 11 days, ducks drinking saltrwater had lost more weight and had higher plasma Na and uric acid concentration and osmolalities than birds drinking freshwater.&lt;/p&gt;&lt;p&gt;3. Saltwater treatment stimulated the salt gland to increased weight and Na, K-ATPase activity.&lt;/p&gt;&lt;p&gt;4. Fenthion generally reduced plasma and brain cholinesterase activity and depressed cholinesterase and Na, K-ATPase activities in salt glands of birds drinking saltwater.&lt;/p&gt;&lt;p&gt;&lt;br&gt;&lt;/p&gt;&lt;p&gt;&lt;/p&gt;</dc:description>
  <dc:format>application/pdf</dc:format>
  <dc:identifier>10.1016/0306-4492(82)90175-7</dc:identifier>
  <dc:language>en</dc:language>
  <dc:publisher>Elsevier</dc:publisher>
  <dc:title>Organophosphate inhibition of avian salt gland Na, K-ATPase activity</dc:title>
  <dc:type>article</dc:type>
</oai_dc:dc>