<?xml version='1.0' encoding='utf-8'?>
<oai_dc:dc xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:oai_dc="http://www.openarchives.org/OAI/2.0/oai_dc/" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xsi:schemaLocation="http://www.openarchives.org/OAI/2.0/oai_dc/ http://www.openarchives.org/OAI/2.0/oai_dc.xsd">
  <dc:contributor>J. H. Soares Jr.</dc:contributor>
  <dc:creator>E. F. Hill</dc:creator>
  <dc:date>1987</dc:date>
  <dc:description>&lt;p&gt;&lt;span&gt;The lethal toxicity of inorganic (HgCl&lt;/span&gt;&lt;i&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;sub&gt;2&lt;/sub&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;/i&gt;&lt;span&gt;) and organic (CH&lt;/span&gt;&lt;i&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;sub&gt;3&lt;/sub&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;/i&gt;&lt;span&gt;HgCl) mercury chloride was compared for&amp;nbsp;&lt;/span&gt;&lt;i&gt;Coturnix&lt;/i&gt;&lt;span&gt;&amp;nbsp;(Japanese quail,&amp;nbsp;&lt;/span&gt;&lt;i&gt;Coturnix&lt;/i&gt;&lt;span&gt;&amp;nbsp;japonica) of different ages from hatch through adulthood by single‐dose acute oral and intramuscular injections and by a 5‐d dietary trial. Sublethal mercury toxicity was studied by evaluation of plasma and brain cholinesterase activity. CH&lt;/span&gt;&lt;i&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;sub&gt;3&lt;/sub&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;/i&gt;&lt;span&gt;HgCl was more toxic than HgCl&lt;/span&gt;&lt;i&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;sub&gt;2&lt;/sub&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;/i&gt;&lt;span&gt;in all tests at each age tested. LD50s consistently increased over the first 4 wk for both acute methods and both mercurials and then stabilized. The striking difference between single‐dose acute and 5‐d dietary tests was that CH&lt;/span&gt;&lt;i&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;sub&gt;3&lt;/sub&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;/i&gt;&lt;span&gt;HgCl averaged about twice as toxic as HgCl&lt;/span&gt;&lt;i&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;sub&gt;2&lt;/sub&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;/i&gt;&lt;span&gt;by both acute methods, compared to 100 times as toxic by the dietary method. For example, at 2 wk of age, the oral LD50s for CH&lt;/span&gt;&lt;i&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;sub&gt;3&lt;/sub&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;/i&gt;&lt;span&gt;HgCl and HgCl&lt;/span&gt;&lt;i&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;sub&gt;2&lt;/sub&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;/i&gt;&lt;span&gt;were 18 and 42 mg/kg and the dietary LC50s were 47 and 5086 ppm. When birds were fed HgCl&lt;/span&gt;&lt;i&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;sub&gt;2&lt;/sub&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;/i&gt;&lt;span&gt;and developed clinical signs of intoxication, they could recover once treatment was withdrawn; however, on CH&lt;/span&gt;&lt;i&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;sub&gt;3&lt;/sub&gt;&lt;span&gt;&amp;nbsp;&lt;/span&gt;&lt;/i&gt;&lt;span&gt;HgCl, clinical signs often commenced after treatment was withdrawn, and then actually intensified for several days and culminated in death.&lt;/span&gt;&lt;/p&gt;</dc:description>
  <dc:format>application/pdf</dc:format>
  <dc:identifier>10.1080/15287398709530964</dc:identifier>
  <dc:language>en</dc:language>
  <dc:publisher>Taylor and Francis</dc:publisher>
  <dc:title>Oral and intramuscular toxicity of inorganic and organic mercury chloride to growing quail</dc:title>
  <dc:type>article</dc:type>
</oai_dc:dc>