<?xml version='1.0' encoding='utf-8'?>
<oai_dc:dc xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:oai_dc="http://www.openarchives.org/OAI/2.0/oai_dc/" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xsi:schemaLocation="http://www.openarchives.org/OAI/2.0/oai_dc/ http://www.openarchives.org/OAI/2.0/oai_dc.xsd">
  <dc:creator>F. F. Fish</dc:creator>
  <dc:date>1934</dc:date>
  <dc:description>&lt;p&gt;1. A fungus disease of epidemic proportions was found in the common sea herring (Clupea harengus) throughout the Gulf of Maine.&lt;/p&gt;
&lt;p&gt;2. The fungus was also found to infect the common winter flounder (Pseudopleuronectes americanus) and the alewife (Pomobolus pseudoharengus).&lt;/p&gt;
&lt;p&gt;3. The causative agent was found to be a species of fungus belonging to the genus Ichthyosporidium Caullery and Mesnil (1905). The specific name is tentatively accepted as hoferi Plehn and Muslow (1911).&lt;/p&gt;
&lt;p&gt;4. The organism is believed to be a normal parasite to the herring and reaches epidemic proportions only when certain unknown factors are operative.&lt;/p&gt;
&lt;p&gt;5. The causative organism was found in herring preserved in 1926, and it is believed that the epidemic has been increasing in severity since that time.&lt;/p&gt;
&lt;p&gt;6. It is believed that such an epidermic,once initiated, increases in severity, reaches a peak, and subsides to a subpatent level. The peak is believed to have been reached in 1931.&lt;/p&gt;
&lt;p&gt;7. The life history and effects of the organism in the herring and flounder are described.&lt;/p&gt;
&lt;p&gt;8. The herring is believed to acquire the infection by the ingestion of parasites liberated from fish in the same school.&lt;/p&gt;
&lt;p&gt;9. The flounder is believed to acquire the infection by the consumption of infected herring.&lt;/p&gt;
&lt;p&gt;10. The alewife is believed to acquire the infection by ingestion of the parasite during its infrequent association with the herring.&lt;/p&gt;
&lt;p&gt;11. Infection is believed to be established by way of the alimentary canal and, once established, to spread throughout the host by way of the blood stream or the lymphatics.&lt;/p&gt;
&lt;p&gt;12. Direct cross infection from the herring to the flounder establishes the theory that the parasites in these two hosts are one and the same organism.&lt;/p&gt;
&lt;p&gt;13. Direct cross infection experiments from the herring to the flounder eliminate the necessity of an intermediate host.&lt;/p&gt;
&lt;p&gt;14. There is no reason to believe that this parasite is capable of infecting warm-blooded animals.&lt;/p&gt;</dc:description>
  <dc:format>application/pdf</dc:format>
  <dc:identifier>10.1017/S003118200002326X</dc:identifier>
  <dc:language>en</dc:language>
  <dc:publisher>Cambridge University Press</dc:publisher>
  <dc:title>A fungus disease in fishes of the Gulf of Maine</dc:title>
  <dc:type>article</dc:type>
</oai_dc:dc>