<?xml version='1.0' encoding='utf-8'?>
<oai_dc:dc xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:oai_dc="http://www.openarchives.org/OAI/2.0/oai_dc/" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xsi:schemaLocation="http://www.openarchives.org/OAI/2.0/oai_dc/ http://www.openarchives.org/OAI/2.0/oai_dc.xsd">
  <dc:contributor>Nancy C. Nelson</dc:contributor>
  <dc:contributor>T. Yasutake</dc:contributor>
  <dc:creator>Gary A. Wedemeyer</dc:creator>
  <dc:date>1979</dc:date>
  <dc:description>&lt;p&gt;&lt;span&gt;Ozone and chlorine inactivation curves were determined in three types of freshwater at 20 C for the destruction of the fish pathogens&amp;nbsp;&lt;/span&gt;&lt;i&gt;Aeromonas salmonicida&lt;/i&gt;&lt;span&gt;&amp;nbsp;the etiologic agent of furunculosis, and&amp;nbsp;&lt;/span&gt;&lt;i&gt;Yersinia ruckeri&lt;/i&gt;&lt;span&gt;&amp;nbsp;the enteric redmouth bacterium (ERM). Ozone and chlorine inactivation curves were also obtained in the same water types at 10 C for the fish pathogenic viruses infectious hematopoietic necrosis (IHNV), and infectious pancreatic necrosis (IPNV). Acute toxicity tests using the rainbow trout as a representative salmonid revealed that ozone was highly toxic at the dose levels used. Partial chronic (3. mo.) testing revealed that ozone exposure at 2 &amp;mu;g/L causes only minimal physiological changes, none of which would be expected to compromise biological function.&lt;/span&gt;&lt;/p&gt;</dc:description>
  <dc:format>application/pdf</dc:format>
  <dc:identifier>10.1080/01919512.1979.10684566</dc:identifier>
  <dc:language>en</dc:language>
  <dc:publisher>Taylor &amp; Francis</dc:publisher>
  <dc:title>Potentials and limits for the use of ozone as a fish disease control agent</dc:title>
  <dc:type>article</dc:type>
</oai_dc:dc>