Marsha L. Landolt Diane G. Elliott Ronald J. Pascho Ronald W. Hardy Ragnar Thorarinsson 1994 <p><span>Groups of juvenile spring chinook salmon naturally infected with </span><i>Renibacterium salmoninarum</i><span>, the causative agent of bacterial kidney disease, were fed diets containing different levels of vitamin E and selenium for 214 days in fresh water and 110 days in seawater. The fish were fed vitamin E at concentrations of either 53±3 mg (designated </span><i>e</i><span>) or 299±9 mg (designated </span><i>E</i><span>) </span><i>α</i><span>-tocopheryl acetate equivalence/kg dry diet in combination with sodium selenite to give selenium concentrations of either 0.038±0.008 mg (designated </span><i>s</i><span>) or 2.49±0.15 mg (designated </span><i>S</i><span>)/kg dry diet. No mortality occurred in the group fed the </span><span id="mmlsi1" class="mathmlsrc"><img class="imgLazyJSB inlineImage" title="Full-size image (<1 K)" src="http://ars.els-cdn.com/content/image/1-s2.0-0044848694902690-si1.gif" alt="Full-size image (<1 K)" width="32" height="30" data-inlimgeid="1-s2.0-0044848694902690-si1.gif" data-loaded="true" data-mce-src="http://ars.els-cdn.com/content/image/1-s2.0-0044848694902690-si1.gif"></span><span> diet, whereas mortality was 3% in the groups fed the </span><span id="mmlsi2" class="mathmlsrc"><img class="imgLazyJSB inlineImage" title="Full-size image (<1 K)" src="http://ars.els-cdn.com/content/image/1-s2.0-0044848694902690-si2.gif" alt="Full-size image (<1 K)" width="31" height="30" data-inlimgeid="1-s2.0-0044848694902690-si2.gif" data-loaded="true" data-mce-src="http://ars.els-cdn.com/content/image/1-s2.0-0044848694902690-si2.gif"></span><span> and </span><span id="mmlsi3" class="mathmlsrc"><img class="imgLazyJSB inlineImage" title="Full-size image (<1 K)" src="http://ars.els-cdn.com/content/image/1-s2.0-0044848694902690-si3.gif" alt="Full-size image (<1 K)" width="28" height="30" data-inlimgeid="1-s2.0-0044848694902690-si3.gif" data-loaded="true" data-mce-src="http://ars.els-cdn.com/content/image/1-s2.0-0044848694902690-si3.gif"></span><span>diets, and 31% in the group fed the </span><span id="mmlsi4" class="mathmlsrc"><img class="imgLazyJSB inlineImage" title="Full-size image (<1 K)" src="http://ars.els-cdn.com/content/image/1-s2.0-0044848694902690-si4.gif" alt="Full-size image (<1 K)" width="27" height="30" data-inlimgeid="1-s2.0-0044848694902690-si4.gif" data-loaded="true" data-mce-src="http://ars.els-cdn.com/content/image/1-s2.0-0044848694902690-si4.gif"></span><span> diet. At the end of the experiment, weight gain and hematocrit values were significantly greater in those fish fed the </span><i>E</i><span> diets compared with those fed the </span><i>e</i><span> diets, whereas the hepato-somatic index was significantly higher in fish fed the </span><i>e</i><span> diets. Glutathione peroxidase activity in blood plasma was significantly higher in fish fed the </span><i>S</i><span> diets compared with those fed the </span><i>s</i><span> diets. No definite effect of dietary vitamin E and selenium on the prevalence and severity of natural </span><i>R. salmoninarum</i><span> infections was demonstrated.</span></p> application/pdf 10.1016/0044-8486(94)90269-0 en Elsevier Effect of dietary vitamin E and selenium on growth, survival and the prevalence of Renibacterium salmoninarum infection in chinook salmon (Oncorhynchus tshawytscha) article